¿Cómo curar la tiroides naturalmente?

¿Cómo curar la tiroides naturalmente? Basado en evidencia científica actual…

Podemos mejorar la función de nuestra tiroides si tomamos en cuenta las siguientes recomendaciones. Funciona para hiper e hipotiroidismo.

Este post se basa principalmente en cambios dietéticos que mejoren la función de tu tiroides y te ayuden a curarla. Sin embargo, la tiroides es un órgano muy complejo y se ve afectado por todo lo que hacemos. Por lo que empezaremos con recomendaciones básicas generales:

1. Dormir más de 5 y menos de 8 horas.
2. Hacer un mínimo de 30 minutos de actividad física al día (sin contar el caminar cuando nos dirigimos a un lugar o cita).
3. Comer de 3 a 5 veces al día, evitando comidas procesadas.
4. Evitar el estrés excesivo, practicar yoga o meditación. El estrés aumenta los niveles de cortisol, lo que altera la función tiroidea
5. Verifica que tus niveles de Yodo, Magnesio, Zinc, Selenio y Manganesio sean los correctos, ya que ellos cumplen un papel fundamental en la función tiroidea.

Ahora sí, vamos a lo de fondo, hay sin fin de testimonios de personas que se han curado siguiendo uno o varios de los siguientes pasos, el que tiene más impacto y evidencia científica es el primero:

1. Dieta vegana o por lo menos una vegetariana muy baja en proteína animal, es decir, mientras menos carnes (incluído pollo y pescado), huevo y leche y derivados mejor.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4377303/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3847753/

2. Dieta libre de soya y maíz GMO (geneticamente modificados), principalmente debido a los pesticidas utilizados, como roundup.

3. Debes comer suficientes frutas y verduras para asegurar los niveles de vitaminas A, complejo B y C que también afectan la función tiroidea.

4. No recomiendo a personas con problemas tiroideos, seguir dietas altas en proteínas como la dieta Atkins.
https://www.ncbi.nlm.nih.gov/pubmed/17341553

5. En algunos casos puede ayudar una dieta libre de gluten (principalmente dejar de consumir cualquier producto que contenga harina de trigo).
https://www.ncbi.nlm.nih.gov/pubmed/18255352

Alimentos que recomiendo, altos en Yodo: arándanos, fresas, habichuelas, frijoles blancos, papa, legumbres en general. El palillo y la maca pueden ser de ayuda, pero sólo añadirlos a tu dieta no tiene el impacto que tiene sí el cambiar tu dieta en los puntos mencionados anteriormente.

*Se puede comer crucíferas, la coliflor, el rábano, entre otros, pero no en exceso

El artículo que acabas de leer es producto de muchas horas de lectura e investigación, por eso me motiva que estés interesado en entender y aprender más sobre la base científica de lo que acabas de leer. Lamentablemente está en inglés pero lo puedes traducir en google translate para tener una idea de que trata cada cosa
Empezemos con las proteínas de origen animal y como una dieta vegana ayuda a regular la tiroides

Postulo que el mecanismo fisiopatológico de como la proteína animal afecta nuestra tiroides está en gran parte relacionado con el aumento de cortisol generado por esta dieta:
https://www.ncbi.nlm.nih.gov/pubmed/10204975
Increased Salivary Cortisol Reliably Induced by a Protein-Rich Midday Meal
“An acute meal-dependent increase in salivary cortisol occurred, which was reliable over 2 test days. This increase in cortisol depended on the proportion of protein in the meal, increasing after the high-protein but not the low-protein meal.”
https://www.ncbi.nlm.nih.gov/pubmed/6270500
Meal Stimulation of Cortisol Secretion A Protein Induced Effect
“Subjects fed high protein diet (4 gm/kg body weight) showed significant increases in cortisol both at 30 and 60 min after the 1200 hr meal and 30 min after the 1600 hr meal. Increases in cortisol, of a smaller magnitude, were also seen after both the 1200 and 1600 hr meals in each of the diets with 1 gm protein/kg body weight (standard, high fat, high carbohydrate). ACTH was significantly increased following the 1200 hr and 1600 hr meals with the high protein diet. We conclude that dietary protein plays an important role in meal stimulated cortisol release.”
https://www.ncbi.nlm.nih.gov/pubmed/3573976
DIET-HORMONE INTERACTIONS PROTEINCARBOHYDRATE RATIO ALTERS RECIPROCALLY THE PLASMA LEVELS OF TESTOSTERONE AND CORTISOL AND THEIR RESPECTIVE BINDING GLOBULINS IN MAN
“By contrast, cortisol concentrations were consistently lower during the high carbohydrate diet than during the high protein diet (7.74 +/- 0.71 micrograms/dl vs. 10.6 +/- 0.4 micrograms/dl respectively, p less than 0.05), and there were parallel changes in corticosteroid binding globulin concentrations (635 +/- 60 nmol/l vs. 754 +/- 31 nmol/l respectively, p less than 0.05).”
https://www.ncbi.nlm.nih.gov/pubmed/21304815
Influence of consumption of a high-protein vs. high-carbohydrate meal on the physiological cortisol and psychological mood response in men and women
“Consumption of the test-meals increased cortisol levels in men in all conditions (p < 0.01), and in women in the rest-protein and stress-protein condition (p < 0.03).”

El incremento de proteína animal puede incluso alterar la concentración de cortisol en los hijos:
https://www.ncbi.nlm.nih.gov/pubmed/12915635
Maternal Consumption of a High-Meat, Low Carbohydrate Diet in Late Pregnancy Relation to Adult Cortisol Concentrations in the Offspring
Nonetheless, our findings provide the first human evidence that an unbalanced high-meat/fish, low-green-vegetable diet during pregnancy may present a metabolic stress to the mother and program the HPA axis of the offspring, leading to lifelong hypercortisolemia. Further studies are needed to replicate the findings and to identify
the nutrient or combination of nutrients responsible for the effect.
https://www.ncbi.nlm.nih.gov/pubmed/17341553
Stress Responsiveness in Adult Life Influence of Mother’s Diet in Late Pregnancy
“The offspring of women who reported greater consumption of meat and fish in the second half of pregnancy had higher cortisol concentrations during the Trier Test. Compared with the offspring of mothers who had reported eating no more than 13 meat/fish portions per week, the average cortisol concentrations were raised by 22% (95% confidence interval, 13 to 71%) and 46% (5 to 103%) in the offspring of those eating 14-16 and at least 17 portions per week, respectively.”

Todo médico sabe que el aumento de cortisol genera una disminución de TSH, la hormona que induce a la formación de más T4, la que posteriormente se convierte en T3 (la forma más activa). Pero los niveles de TSH podrían afectar también los niveles de cortisol. Es decir, cuando aumentas tus niveles de cortisol afectas a la tiroides, pero una tiroides afectada puede afectar también a los niveles de cortisol y generar un proceso crónico:
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3520819/
Elevated thyroid stimulating hormone is associated with elevated cortisol in healthy young men and women.
“Results suggest a positive relationship between TSH and cortisol in apparently healthy young individuals. In as much as this relationship may herald a pathologic disorder, these preliminary results suggest that TSH levels > 2.0 uIU/L may be abnormal. Future research should address this hypothesis further, for instance through an intervention study.”

Conexión entre función tiroidea y enfermedad celíaca, o intolerancia al gluten:
https://www.ncbi.nlm.nih.gov/pubmed/9872614
Autoimmune thyroid diseases and coeliac disease
“The association of coeliac disease with autoimmune thyroid disease is not surprising as they share common immunopathogenetic mechanisms. It is advisable to screen autoimmune thyroid disease patients for coeliac disease as there is an increased risk for gluten intolerance.”
https://www.ncbi.nlm.nih.gov/pubmed/15244201
The presence of the antigliadin antibodies in autoimmune thyroid diseases
“We claim that polyglandular endocrine syndrome is the commonest cause of positivity of antigliadin antibodies in the patients with autoimmune thyroid disease.”
https://www.ncbi.nlm.nih.gov/pubmed/11768252
Clinical and subclinical autoimmune thyroid disease in adult celiac disease
“In conclusion, the occurrence of both clinical and subclinical autoimmune thyroid disease was increased in celiac disease; subclinical thyroid disease indicates regular surveillance.”
https://sma.org/…/celiac-disease-occurring-in-a-patient-wi…/
“Patients with one autoimmune endocrine disorder have an increased risk of developing other autoimmune diseases.”
“The association between autoimmune thyroid disease and celiac disease is common.”
https://www.ncbi.nlm.nih.gov/pubmed/8130887
Autoimmune thyroid disorders and coeliac disease
“All newly detected coeliac patients had IgA-class gliadin, reticulin and endomysium antibodies, but none of the patients had any gastrointestinal symptoms or abnormal biochemical findings suggesting coeliac disease. Treatment of thyroid disorders and coeliac disease was successful in these patients. The present results confirm that the frequency of subclinical coeliac disease is increased among patients with autoimmune thyroid disorders. IgA-class reticulin, endomysium or gliadin antibody tests are suitable screening methods for detecting these patients, as far as selective IgA-deficiency is excluded.”
https://www.ncbi.nlm.nih.gov/pubmed/25925917
Celiac Disease and Endocrine Autoimmunity
“The genes that predispose to endocrine autoimmune diseases, e.g. type 1 diabetes, autoimmune thyroid diseases, and Addison’s disease, i.e. DR3-DQ2 and DR4-DQ8, are also the major genetic determinants of CD, which is the best understood HLA-linked disease.”
https://www.ncbi.nlm.nih.gov/pubmed/17075442
Screening for celiac disease in Tunisian patients with Graves’ disease using anti-endomysium and anti-tissue transglutaminase antibodies
“Patients with Graves’ disease are at substantial risk of CD and therefore antibody screening for CD may be included in the work-up of these patients. Either AEA or AtTG may be used.”
https://www.ncbi.nlm.nih.gov/pubmed/15730411
Prospective screening for coeliac disease in patients with Graves’ hyperthyroidism using anti-gliadin and tissue transglutaminase antibodies
“Screening 111 consecutive patients with Graves’ hyperthyroidism revealed AGA in 14%, anti-tTG in 2% and IgA deficiency in 3%. Two patients were known to have CD. Screening detected three new cases. The prevalence of CD in patients with Graves’ hyperthyroidism was 4.5% as compared with 0.9% in matched healthy controls. Routine screening for CD should be considered.”

Como el consumo de gluten puede generar problemas de absorción y mecanismos que alteran la función tiroidea:
https://www.ncbi.nlm.nih.gov/pubmed/12625811
IgA and IgG Antigliadin, IgA Anti-tissue Transglutaminase and Antiendomysial Antibodies in Patients with Autoimmune Thyroid Diseases and Their Relationship to Thyroidal Replacement Therapy
“Our results confirm that patients with higher thyroid replacement requirements also have higher antibody titres, especially AGA-IgA. On the basis of this fact, we can draw two conclusions: 1) impaired intestinal immunity can lead to the initiation or progression of thyroidal autoimmune disorders with low production of hormones, and 2) an impaired resorption of various substances may occur (for levothyroxine in persons undergoing replacement therapy, for iodine, selenium, calcium, vitamin D, iron etc).”
https://www.ncbi.nlm.nih.gov/pubmed/19014325
Tissue Transglutaminase Antibodies in Individuals with Celiac Disease Bind to Thyroid Follicles and Extracellular Matrix and May Contribute to Thyroid Dysfunction
“Our study supports a role for anti-TGase II antibodies as a contributing factor in the development of thyroid autoimmunity. This, together with epidemiological data demonstrating a strong association of celiac disease and autoimmune thyroid disease (52), suggests that those with celiac disease should be screened for autoimmune thyroid disease.”
https://www.ncbi.nlm.nih.gov/pubmed/10931424
Gluten-dependent diabetes-related and thyroid related autoantibodies in patients with celiac disease
“In conclusion, this study on IDDM and thyroid-related autoantibodies in children with CD provides laboratory data in support of the hypothesis that a GFD started early may prevent the other autoimmune diseases frequently associated with CD. Assuming that organ-specific autoantibodies do indeed play either a predictive pathogenic or diagnostic role in the development of those autoimmune diseases frequently associated with CD and that a GFD started as early as possible may prevent the onset of these disorders, it is conceivable that 100% of patients with CD and associated IDDM or auto-immune thyroiditis should be positive for the corresponding class of autobodies. It is noteworthy that in our own clinical experience, all of the 28 patients with CD and associated IDDM tested positive for IDDMrelated autoantibodies (unpublished findings), and that all 6 patients with CD and associated thyroiditis tested positive for serum TPO antibodies.”
https://www.ncbi.nlm.nih.gov/pubmed/10419909
Duration of Exposure to Gluten and Risk for Autoimmune Disorders in Patients With Celiac Disease
“In conclusion, our data for the first time strongly suggest that the prevalence of autoimmune disorders in celiac disease depends on duration of exposure to gluten.”
http://www.thelancet.com/…/PIIS0140-6736(05)64411-X/abstract
Coeliac disease and autoimmunity due to unmasking of cryptic epitopes
“It is now tempting to speculate that digested gliadin, known to have high affinity for jejunal mucosal lamina propria reticulin, unmasks cryptic reticulin/endomysial epitopes, the CDAP, as recently suggested. 2 Specific HLA class II molecules on antigen-presenting cells could present the self-peptides, thus activating the autoreactive T-cell population. In fact, the immune response to the partly purified protein complex Fb-CDAP is strictly regulated by the alleles DQA1*0501 and DQB1*0201 and can be generated in vitro in genetically predisposed persons in the absence of coeliac disease. 6 Continuing gliadin ingestion (analogous to recurrent “infection”) may be responsible for the self-maintenance of the disease. The autoaggressive T-cell population should be identified.”

Algunos casos sobre regulación de la función tiroidea con dieta libre de gluten:
https://www.ncbi.nlm.nih.gov/pubmed/25325278
Celiac Disease in a Predisposed Subject (HLA-DQ2.5) with Coexisting Graves’ Disease
“A 47-year-old woman complained of persistent chronic diarrhea and weight loss over a 9 month period. Results of all serological tests and stool exams were negative. However, the patient was found to carry the HLA DQ2 heterodimer. Symptoms improved after a gluten-free diet was initiated. The patient has been followed and has suffered no recurrence of symptoms while on the gluten-free diet. An overall diagnosis of celiac disease was made in a genetically predisposed patient (HLA-DQ2 heterodimer) with Graves’ disease.”
https://www.ncbi.nlm.nih.gov/pubmed/25342186
Graves’ disease and coeliac disease screening and treatment dilemmas
“We report a case of a 37-year-old woman with Graves’ disease (GD) who presented with thyrotoxicosis that was not responsive to medical management. The screening for coeliac autoimmunity (CA) was positive. After initiation of a gluten-free diet the patient’s thyrotoxicosis responded to medical management.”

Dietas altas en proteínas como la Atkins son malas para las personas con alteración de la función tiroidea, afectando incluso a los hijos de los que utilizan estas dietas:
https://www.ncbi.nlm.nih.gov/pubmed/17341553
Stress Responsiveness in Adult Life Influence of Mother’s Diet in Late Pregnancy
“A high-protein diet is known to stimulate the HPA axis, possibly by means of the direct neuroendocrine effects of ingested neurotransmitter substrates including l-tyrosine and l-tryptophan.”
“Nevertheless, high-protein diets, such as the Atkins diet, although not recommended during pregnancy, are popular in young women today, and this study raises the possibility that they may have long-term health effects on the offspring.”

También hay artículos como este, que contradicen el beneficio de una dieta libre de gluten, pero hay que tener en cuenta que de nada sirve tal vez reducir el guten de la dieta, si eso genera un aumento del consumo de proteína animal, y eso puede generar resultados como estos:
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2111403/
Celiac Disease and Autoimmune Thyroid Disease
“Patients with CD may present with diverse clinical manifestations, or without symptoms, to specialists other than gastroenterologists. There is ample evidence of a strong association between CD and several immune mediated diseases, including autoimmune thyroid disorders, type 1 diabetes mellitus, primary biliary cirrhosis, inflammatory bowel diseases and autoimmune adrenal failure. Some of these conditions share HLA haplotypes and non-HLA alleles, e.g., CTLA-4, which may underlie their pathogenesis.
Thyroid function should be assessed in all CD patients at diagnosis and follow-up if clinically indicated.
Currently there is little evidence to support a role for a gluten-free diet in reducing the development of associated autoimmune disorders in patients with CD, but early diagnosis and dietary treatment reduces complications such as malabsorption, osteoporosis and lymphoma, and improves the absorption of drugs.”

Si te pareció suficiente evidencia y conoces a alguien con problemas de la tiroides no olvides de compartir la información
Si tienes alguna duda o sugerencia puedes escribirme un mensage a esta página.
Que tengas buen día.

Exoneración de responsabilidades: El presente artículo presenta opiniones e ideas de su autor. Y tiene como propósito difundir información general sobre los temas tratados y ayudar a los lectores a tomar decisiones informadas sobre su salud. No es de modo alguno un sustituto para las indicaciones del médico tratante del lector, que da un tratamiento personalizado, en base a los síntomas y condiciones individuales de cada paciente. Si el lector necesita ayuda médica, debe consultar un médico capacitado primero.

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